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   sci.med.psychobiology      Dialog and news in psychiatry and psycho      4,734 messages   

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   Could coconut oil help fight ageing? Hig   
   26 Nov 14 16:36:45   
   
   From: 23x11.5c@gmail.com   
      
   Could coconut oil help fight ageing? High-fat diet could halt Alzheimer's and   
   Parkinson's, study finds   
      
   Researchers analysed mice with a defect in their DNA repair system   
   In humans, this defect causes Cockayne syndrome: premature ageing   
   High fat diet postponed signs of ageing - impaired hearing or weight loss   
   Also postponed ageing of the brain and could stave off disease   
   Experts said fats gave the brain enough fuel to repair cell damage    
   Ageing is caused when cells and DNA become damaged    
   Hopes the discovery could pave the way for new treatments for patients with   
   Alzheimer's and Parkinson's disease    
      
   By MADLEN DAVIES FOR MAILONLINE   
      
   PUBLISHED: 12:19 EST, 24 November 2014   
   UPDATED: 14:31 EST, 24 November 2014   
      
      
   Eating a diet high in fat could stave off Alzheimer's and Parkinson's disease   
   and other signs of ageing, new research claims.   
      
   High-fat diets postponed signs of ageing including impaired hearing and weight   
   loss, and also stopped ageing of the brain, a study found.   
      
   Scientists discovered medium chain fatty acids, like coconut oil, were   
   particularly effective.     
      
   They believe the fats help slow down the ageing process, by helping to repair   
   cells and DNA damaged as a result.    
      
   Scroll down for video    
      
   Fats such as coconut oils give the brain enough fuel to repair the damage to   
   cells and DNA that causes ageing. Eating a high fat diet postponed signs of   
   ageing including impaired hearing and weight loss   
   Fats such as coconut oils give the brain enough fuel to repair the damage to   
   cells and DNA that causes ageing. Eating a high fat diet postponed signs of   
   ageing including impaired hearing and weight loss   
      
   As part of the new study, researchers analysed mice with a defective DNA   
   repair system.    
      
      
   In humans, this defect causes the disorder Cockayne syndrome, where patients   
   prematurely age as children and have a life-expectancy of between 10 and 12   
   years.   
      
   Researchers found placing a mouse with this defect on a high-fat diet   
   postponed ageing processes such as impaired hearing and weight loss.   
      
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   It also postponed the ageing of the brain, giving hope to children who age   
   prematurely and patients suffering Alzheimer's and Parkinson's disease.   
      
   The study's lead author Professor Vilhelm Bohr, of the University of   
   Copenhagen, said: 'The study is good news for children with Cockayne syndrome,   
   because we do not currently have an effective treatment.   
      
   WHAT IS COCKAYNE SYNDROME?    
   Cockayne syndrome (CS) is very rare disease resulting in slow growth in babies   
   or rapid aging in children.   
      
   Persons with this syndrome have smaller than normal head sizes, are of short   
   stature, their eyes appear sunken, and they have an 'aged' look.   
      
   It is estimated to affect one in 500,000 children but difficult to diagnose.   
      
   For CS patients who are affected at birth, life expectancy is three to seven   
   years.   
      
   Other patients only start to show symptoms in early childhood and their life   
   expectancy tends to be under 20 years.   
      
   However, some rare CS patients live significantly longer.   
      
   Individuals with CS are extremely sensitive to the sunlight and sunburn easily   
   so they mostly stay indoors during normal daylight hours.   
      
   CS is often described as a 'progeria-like syndrome'.    
      
      
      
   'Our study suggests that a high-fat diet can postpone ageing processes. A diet   
   high in fat also seems to postpone the ageing of the brain.   
      
   'The findings therefore potentially imply that patients with Alzheimer's and   
   Parkinson's disease in the long term may benefit from the new knowledge.'   
      
   Researchers explained our brain has a constant need for fuel in the form of   
   either sugar or ketones.   
      
   Ketones are the brain's fuel reserve, and play an important role in periods of   
   low blood sugar levels, such as when a person is fasting.   
      
   This is because the body breaks down fat if it needs sugar, and during this   
   process it produces ketones.   
      
   The researchers saw a particular positive effect when the mice were given   
   medium chain fatty acids - coconut oil, for example.   
      
   Previous research has shown that in children with Cockayne syndrome, ageing is   
   a result of the cell repair mechanism being constantly active.   
      
   This means it eats into the cell's resources and causes it to age very quickly.   
      
   'We therefore hope that a diet with a high content of coconut oil or similar   
   fats will have a beneficial effect, because the brain cells are given extra   
   fuel and thus the strength to repair the damage,' said Morten Sc   
   eibye-Knudsen, from the National    
   Institute of Health, who also carried out the study.   
      
   The research was published in the journal Cell Metabolism.    
      
   The health benefits of coconut oil - and how to use it   
      
    High fat diets also postponed ageing of the brain. Experts said fats such as   
   coconut oils could be used to stave off Alzheimer's and Parkinson's disease   
   (file pic)   
   High fat diets also postponed ageing of the brain. Experts said fats such as   
   coconut oils could be used to stave off Alzheimer's and Parkinson's disease   
   (file pic)   
      
      
      
   http://www.dailymail.co.uk/health/article-2847618/Could-coconut-   
   il-help-fight-ageing-High-fat-diet-halt-Alzheimer-s-Parkinson-s-   
   tudy-finds.html   
      
   --- SoupGate-Win32 v1.05   
    * Origin: you cannot sedate... all the things you hate (1:229/2)   

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