From: jshem@yaxenu.org   
      
   JAB  writes:   
      
   > Alzheimer's as autoimmune disease   
   >   
   > We believe that beta-amyloid is not an abnormally produced protein,   
   > but rather is a normally occurring molecule that is part of the   
   > brain's immune system. It is supposed to be there.   
   >   
   > When brain trauma occurs or when bacteria are present in the brain,   
   > beta-amyloid is a key contributor to the brain's comprehensive immune   
   > response. And this is where the problem begins.   
   >   
   > Because of striking similarities between the fat molecules that make   
   > up both the membranes of bacteria and the membranes of brain cells,   
   > beta-amyloid cannot tell the difference between invading bacteria and   
   > host brain cells, and mistakenly attacks the very brain cells it is   
   > supposed to be protecting.   
   >   
   > This leads to a chronic, progressive loss of brain cell function,   
   > which ultimately culminates in dementia - all because our body's   
   > immune system cannot differentiate between bacteria and brain cells.   
      
   Very interesting, right?  That's the current understanding.  For now,   
   the message seems clear---we need to keep the bacteria out.  How did   
   they get in there in the first place (in the out-of-balance numbers)?   
   Why is the immune system having to handle so much bacteria in the brain?   
      
   --- SoupGate-Win32 v1.05   
    * Origin: you cannot sedate... all the things you hate (1:229/2)