From: treon3verdery@gmail.com   
      
   two approaches to this periodicity of administration of the mRNA bremelanotide   
   causes the production of are a pill, iontophoretic dermal applique, or nasal   
   spray that contains a bremelanotide and flibanserin associated mRNA in a   
   plural enteric    
   microdissolving time sequenced mini pill like contac™. The bremelanotide   
   mRNA drugs would cause a fresh rapid rush of sexual avidity, erotic visual   
   noticing, and genital awareness at both chromosomal sexes once every 16   
   minutes that lasts 8 minutes.    
   Other drugs besides bremelanotide that cause high sexual avidity, higher   
   frequency of sexual thoughts, and measureably more frequent initiation of sex   
   with a partner exist. Among them 2-CB, cocaine, and methamphetamine are drugs   
   i have heard of    
   Additionally, the brain localization of just the things about cocaine that   
   cause greater sexual avidity, arousal, and initiation of sexual contact would   
   co-occur with the start of the 16 minute mRNA of paleness bremelanotide and   
   mRNA of flibanserin. The    
   brain localization of just the sexy part of cocaine and just the sexual   
   avidity part of aphrodesiac 2-CB drug, and just the sexual avidity part of   
   methamphetamine could be found by linking these drugs to brain localization   
   aptamers that preferentially    
   accumulate at fMRI and positron emission tomography of volunteers with the   
   99.9th percentile of a combination of high voluntary partner sexual activities   
   per 24 hours, high spontaneous incidence frequency, and physical sexual   
   arousal height sexual    
   thoughts, fantasies, and initiations of partner sexual activity at both   
   chromosomally female persons and chromosomally male persons. Noting that these   
   specific brain areas, and possibly pelvic ganglion areas might have only   
   approximate brain localization    
   maps with peptide aptamers, a fresh group of 100K new stable isotope labelled   
   peptide aptamer variations on nearest brain area localization can be tested on   
   paid volunteers to find superspecific brain localization peptides and peptide   
   aptamers to the    
   fMRI and positron emission tomography brain areas of high sexual avidity, and   
   high physiological sexual arousal, and high orgamic intensity and multiplicity   
   of orgasms, as well as brain activation areas of paid vulunteers who   
   immediately want to restart    
   sexual motion and touch after their first orgasm. Similarly 100K new map   
   variants of peptide localization drugs, including peptide aptamer location   
   drugs can be characterized to map to microspecific areas of the pleasure and   
   enjoyment causing parts the    
   sexual orgasm, with or without partner then causes a very large multiminute   
   nucleus accumbens, pallidum, along with other pleasure centers 16 minute rush   
   from each orgasm. Notably rodents produce unique mRNA from orgasm events so it   
   is very likely humans(   
   Homo sapiens and branch species) also activate a specific gene or genes when   
   having an orgasm. Notably this gene expressed during orgasm could be made into   
   a fusion gene that makes a nucleus accumbens, pallidum, and other pleasure   
   center localization    
   peptide, that is linked to another receptor tail nestling, and thus receptor   
   activation causing pleasure, thrill, rush, glow, stimulant effect at the   
   nucleus accumbens and pallidum, and other pleasure centers. This stimulation   
   rush effect at dopamine    
   receptors, trace amine (TAAR) receptors and anandimide receptors extends the   
   pleasure caused by an orgasm with a 16 minute pleasure rush that causes, from   
   dopamine stimulation and viagra like PDE-5 decrease, measurably increased   
   sexual motions,    
   automaticity of sexual motions, and increased clitoral, vaginal and penile   
   tumescence. The effect of orgasm on people with penises is greater immediate   
   and automated movement of continued and continuing sexual contact as well as   
   more intense harder    
   immediate penile rigidity so the person with a penis-in-vagina stays hard   
   after orgasm and continues to thrust, pleasuring the person with a vagina.   
   Causing person with a penis orgasm and ejaculation to immediately cause a   
   continued and increasing penile    
   hardness and rigitity comes from the mRNA produced with intromission of a   
   penis in a vagina and several thrusts (published at rodents) being made part   
   of a fusion gene that produces a penile corpus cavernosum localization peptide   
   linked to an opiate    
   peptide that functions like papaverine to cause immediate increased and   
   sustained penile hardness or also strong sustained clitoral and possibly   
   vaginal tumescence. Before and after orgasm the penis stays as rigid as   
   papaverine can cause, and before and    
   after orgasm the clitoris and vaginal canal, and optimally vaginal distal   
   O-Spot and A-spot are as tumescent as simultaneous papverine functionalike   
   opiate peptide simultaneously experienced with a viagra-like PDE-5 decreaser   
   can cause. At both people    
   with penises and people with a clitoris and vagina, the sustained genital   
   capability and rigidness/turgor that lasts 40 minutes after first orgasm, and   
   the increased automaticity of movement and sexual avidity at all partners from   
   woman on top sex    
   positions, and the increased automaticity of thrusting and sexual avidity of   
   all partners during man on top get rid of what was called the sexual   
   refractory period during the 20th century AD.    
      
      
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   --- SoupGate-Win32 v1.05   
    * Origin: you cannot sedate... all the things you hate (1:229/2)