From: treon3verdery@gmail.com   
      
   veterinary products; noting 3000% markup of human diagnostic tests   
   ebay<->alibaba   
      
   veterinary diagnostics   
      
   wood and lumber and factory made doors and windows and drywall:  pressure   
   treated wood and lumber and factory made doors and windows and drywall could   
   be pressure treated with antimicrobial and antibacterial and antifungal   
   peptides; D amino acid versions    
   might omit enzymatic degradation by bacteria and fungi This   
   Could be particularly value effective as they could use just 10-90% as much of   
   the pressure treated wood chemicals they use now, with something like a   
   micrograms/Kg of lumber dose of peptide; that might be replacing 10c of   
   chemicals with 2/100,000 of 1    
   cent of peptides (100 micrograms/Kg, 20 cents//G; but really at lumber volumes   
   it would likely be less than 1KG of oxytocin at alibaba 20 cents a gram,   
   $200/Kg.  This also brings up the possibility of tuning a lumber   
   antimicrobial/antibacterial/   
   antifungal to be positively or negatively charged, hydrophilic or lipophilic   
   to have greatest cheapest depth of permeation with the most rapid treatment;   
      
   Insects share some percentage of their genes with humans; those genes that   
   only exist at insects, notably insects that colonize/eat wood could have   
   peptide and RNA drugs tailored to be lethal to to the insects; Insects may   
   have completely unique   
      
   farthest from anything in the human genome sequences, and proteome products;   
   99th percentile most foreign to humans genes and proteins as places to   
   terminate insects; RNA and peptide drugs that target these least-human but   
   at-insect could be much less    
   likely to have any human or mammal bioactivity and so are much safer as   
   pesticides.   
      
   silkworms do not have pineal glands (melatonin, epithalon) but live different   
   amounts of time, varying 20-35% based on the photoperiod they live at; mRNA   
   that then finds the genes of greater longevity from (optimal) photoperiod   
   could have human analogous    
   genes, and those human genes could code for circulating longevity chemicals or   
   longevity receptors; they could test gene allele and SNP and epigenetic   
   variations at mice to see if the analogous human and mouse genes to silkworm   
   longevity photoperiod    
   genes cause greater longevity at mice   
       
   ACtually, they could feed silkworms royal jelly to see if they lived longer as   
   well, and then trace that to genes, and then to mouse and humans genes as well.   
      
   longevity technology: other species’ hormesis, where the hormesis is not at   
   mice (or as far as is known humans), the other species hormesis does however   
   upregulate analogous genes at mice and humans;  nonmouse monhuman gene   
   products that are rptective    
   or longevizing in other species hormeisis could, wehn injected or fed to mice,   
   cause greater wellness or longevity; So, for example, insect hormesis   
   hemolymphy circulating factors, or possibly completely different HSP/CSP than   
   mammals make could be    
   beneficial to mammals like humans; extreme radiation hormesis at insects (high   
   dose radiation tolerance; also silkworms apparently do better with 254 nm UV;   
   so rather than just say “radiation hormesis, perhaps testing 11-80 different   
   spectral bands of    
   radiation from ThZ to gamma radiation, and nonthermal ultra high energy radio   
   (AM/FM/WIFI/phone) to see if they have unique genes and mechanisms, and, for   
   longevity technology and wellness technology, different hormesis gene   
   activations and gene products;   
      
   Longevity technology:  Things that make lungfishes live longer could activate   
   completely unique longevity genes, and those gene products as circulatory   
   circulating chemicals could also effect mice and humans as longevity drugs;   
   wikipedia says the    
   lungfish has the very largest genome of any animal (133 billion base pairs;   
   humans 3 billion base pairs); so the 44+ times more genes it has than a human,   
   could be a protein product space 44 times larger than anything naturally made   
   at humans, these (   
   over simplistically enumerted; it’s more at proteome and peptide-ome)   
   4,400,000-or more unique lungfish gene products (human 100K genes with open   
   reading frames; 100K gene products; 4.4 million gene products at lungfish   
   genome with open reading frames)    
      
   So, things that make lungfish libe longer have a 44:1 likeliness of being new   
   unique.  so just breeding lungfish to live 10-100% longer, as has been done   
   with drosophila and c elegans, produces large numbers of new longevity genes   
   and gene products.   
       
      
   hyperfasting; n>10 million insects, such as genetically diversified and   
   radiation exposed silkworm egg-layers progeny insects; 99.9th percentile of   
   fasting survivors is 100 particular clonable, rebreedable insects to find any   
   additional xeno-to-human    
   protein products associated from fasting; considering fasting as hormesis,   
   other forms of hormesis could be tried on 1 million silkwork colonies   
   addordably, CSP, HSP radiation amounts and frequency bands, ultrasonic   
   disruption of tissue, THz and radio    
   disruption of tissue, carcinogens, stimulants that preclude sleep (I read   
   insects sleep), depressnats that permit food consumption; opiate overdose;   
   overdose of   (hormesis)   
      
   microexamples: opiate peptides at mammals cause reduction in cardiovascular   
   disease and produce/accompany hibernation causing greater lifespan; the 99.9th   
   percentile of insects that live longer even though they are 99% terminated   
   with combinatorial mixed    
   opiate activator proteins, and a group that lives longer than 99% at mixed   
   opiate antagonist peptide each have paarticular chemicals and responses that   
   could cause a mouse they were injected into to live longer by a completely   
   non-opiate peptide    
   mechanism, similarly, mice that live longer on teh insect 99.9th percentile of   
   lifespan increasign opiate peptide antagonist have a new way to live longer   
   that is not sedating.  So, a hibernation-quality longevity peptide, that omits   
   causing hibernation (   
   bodyside only version may be absent deleterious effects)   
      
   Genetically modifying (knocking out) screen 1-10 million insect batches for   
   several known longeveity pathways simultaneously then finding the 99.99   
   percentle of longevity at the survivors find completely unpublished longevity   
   genes (40% analogous genes    
   insects and humans shared) at the 99.9 percentile of silkworm longevity:   
      
   [continued in next message]   
      
   --- SoupGate-Win32 v1.05   
    * Origin: you cannot sedate... all the things you hate (1:229/2)