From: treon3verdery@gmail.com   
      
    screening a million molecular variations on lithocholic acid could create new   
   longevity drugs; at humans, humans that have greater amounts of lithocholic   
   acid have higher cancer risk, I read mice tend to get cancer more than humans   
   so when screening the    
   99.99th percentile of the most longevizing molecular variants of lithochilic   
   acid it is possible a mammal lthat has less cancer risk than mice, as well as   
   age batched marmosets could be a better model for quantifying longevity   
   increase; an online item    
   says lithocholic acid is toxic but not at humans when medically adminsteref,   
   it is also possible that at humans the coadministration of cancer risk   
   reducing longevity chemical like metformin or rapamycin could cause the actual   
   cancer risk of rabbits as    
   well as mice as well as marmosets to be less that that of completely   
   unmedicated rabbits, mice, or marmosets; cholesterol risks could be reduced to   
   less than those of unmedicated mammals with the high density lipoprotein   
   cholesterol reducing drug    
   simvastatin which is published as having a longevity benefit, although   
   possibly from heightening healthspan   
      
   Alibaba lithocholic acid dose; at humans it is published that 30mg/24 hours of   
   ursolithocholic acid causes 16.86 micromolar concentration ( up from .05), and   
   bile acid amount went up to 17.21 (all the bile acids) suggesting that 50   
   micromolar    
   concentration like the yeast that live twice as long is a 89 mg/24 hours;   
   although it might not be a plausible dose: the paper says 30 mg of   
   ursolithocholic acid causes only .22 micromolar concentration of lithocholic   
   acid at humans; perhaps the 17.21    
   millimolar concentration of all kinds of bile acids, 6 of which make yeast   
   live longer, compensates for that; another paper says ursodeoxycholic acid (a   
   hydrophilic bile acid at ( 30mg/kg each 24 hours) makes previously unwell   
   humans 2.1times more likely    
   to omit being alive than unmedicated humans, note though that that is at 30   
   mg/kg/24 hours so is 70 times higher than the 30 mg makes 17 micromolar   
   concentration at humans so it is possible that a 1/70th dose has only a 1.03   
   (compared with one)    
   likeliness of causing not being alive;    
      
   it is possible that doing correlative studies of human longevity from the   
   varying amounts of bile acid that occur at the different phenotypes at the   
   human population could find genes that make concentrations of bile acid   
   molecules different than others,    
   if there are versioms of three or fourteen different kinds of genes that   
   effect bile acid actual different molecule amount then those three or fourteen   
   genetic variations, their SNPs, alleles, and copy numbers could correlate with   
   different human    
   longevity, also they could look at the genetics and measured amounts of bile   
   acids at super centenarians to find out if there is a more longevizing   
   genetics of bile acids or amount of bile acids   
      
      
   I do not know if there is a yeast compensation number when calculating a human   
   dose from a yeast dose like the order of magnitudish less drug compensation   
   number at mice   
      
   Write about lithochilic acid online like longevity.org   
      
   --- SoupGate-Win32 v1.05   
    * Origin: you cannot sedate... all the things you hate (1:229/2)