From: treon3verdery@gmail.com   
      
   from staying awake for 48 hours a few days a week I got mild dermatitis, when   
   I was under 18 I had atopic dermatitis, I noticed then that it was really   
   distracting, to put it diffidently, the distracting part of the symptoms of   
   dermatis nearly completely    
   cease when i take about 2.5 grams of phenibut. And the anti itch cool,   
   original color skin lasts 16 hours;   
      
   An oral anaesthetic that localizes at the dermis or sCNS blocking of nerve   
   communication? skin heals more rapidly when I take phenibut suggesting dermis   
   neuron or other effects; that's wonderful because some kind of molecular   
   activity amplifier like    
   halogen (perhaps gabapentin which is a lot like chlorophenibut) or some new to   
   me thing would be functional;   
      
   Orally dosed body side phenibut, that is a phenibut variant that stays on the   
   body side of the blood brain barrier, could heal dermis while removing almost   
   all symptoms, also dermatitis rash cools and dermis turns to the well white   
   Caucasian  color of    
   the areas without  dermatitis after a few hours to 24 hours, so if a modified   
   phenibut molecule, perhaps with 400 amu moiety on it that keeps it from   
   passing the blood brain barrier cools, and removes the dermatitis rash visual   
   effect as well as causes    
   actual healing with skin softening as rapidly as 24-48 hours without washing   
   or being clean when dirty, then that body side version of phenibut could also   
   be absent what I read about phenibut at the CNS; if you take it frequently   
   like 7 days a week your    
   CNS GABA receptors will deregulate and your undrugged mood might be more   
   restless, kind of like an adaption to sedation even though phenibut is a   
   nootropic, so it is possible body side only phenibut could be utilized 7 days   
   a week without deregulating a    
   person's CNS and effecting their mind; so body side only variation on the   
   phenibut molecule   
      
   Also phenibut could make peripheral nervous system side reflex better   
   (nootropic), or slower (GABA sedative, just at the peripheral nervous system;   
      
   noting 7 days a week of phenibut for a month could deregulate gaba receptors   
   and CNS based emotions, would quitting body side phenibut cause a "rebound   
   rash", at least that is easy to test with just a few N to get a p value;   
   noting continuous multiday    
   dosing a GABA active drug causes receptor change of some kind at the CNS, if   
   there is rebound rash at dermis, even with a bodyside molecular variant that   
   does not reach the CNS, then screening a library of phenibut  molecules is   
   possibly a thing where    
   noting without an absence of "rebound rash" it is not functional, if a new   
   phenibut atopic dermatitis molecule is reboundrasless and functional it   
   becomes a highly wonderful one pill a day atopic dermatis symptom removing   
   pill that also causes healing    
   even without washing, something that when i had atopic deratitis when i was   
   less than 18 bothered me and sometimes washing seemed to make the atopic   
   dermatitis even less pleasant; screening a library of a few hundred, or a   
   million wells at a dermal    
   tissue culture plate could find a phenibut variant of greater effectiveness   
   per mg;  note I do not know if "rebound rash" actually exists   
      
   Dermal localization, 1/40th the dosage; oral phenibut is 2.5 grams for a dose,   
   localizing forms of the molecule could make the oral dose 63 mg, which is   
   beneficial as 2.5g is a few big pills, or alternately, like phenibut, a take   
   with water drug and it    
   tastes crummy;   
      
   Is a body side dermal soothing and healing effect from GABA, or is it some   
   other chemical effect, if it is some other effect then screening a library of   
   orally active phenibut molecule variations that are nongaba active therefore   
   also completely    
   different than gaba active phenibut molecular variants would be beneficial.   
   Radiolabelling phenibut would also locate the nonGABA receptors at the skin   
   that phenibut might be reaching, this could be quantified as to how much  of   
   the radio labelled    
   phenibut is reaching what parts of the dermis and they could make a localizing   
   version that just went to the preferred area of the dermis, optimally just the   
   layers of the dermis where it soothes itching, and the layer where oral   
   phenibut makes healing    
   even more rapid than a person that has not bathed in 22 days washing;   
   Halogened glucocorticoids are a 2019 dermatitis medication, it is possible   
   that attaching just the part of the phenibut molecule to the halogenated   
   glucocorticoid would make a new screenable library to find a beneficial atopic   
   dermatitis treatment;   
   So I read some children got atopic dermatitis during 2019, this technology   
   benefits them;   
   Also halogenated glucocorticoid ointment itches, if phenibut also functions   
   topically this could be an itchless a topic dermatitis treatment   
      
   --- SoupGate-Win32 v1.05   
    * Origin: you cannot sedate... all the things you hate (1:229/2)