From: treon3verdery@gmail.com   
      
    longevizing recreational drug highs to nonhuman mammals, Enzymatically   
   determined dosage of a recreational drug, herbivores might eat kilograms of   
   dry, slightly moldy, recreational drug containing grass during winter, while   
   other mammals seeking an    
   enjoyable recreational drug experience like bears might eat only grams, a way   
   to make it so a plurality, optimally all species are able to enjoy a harmless    
   recreational drug experience even while eating orders of magnitude different   
   amounts of the plant,   
    fungi, hair oil or hair-saliva product, is to have the drug be a drug that   
   only turns active when liver enzymes activate it, the first grams of what is   
   consumed utilize all of the amount of that particular liver enzyme, then the   
   rest of the drug,    
   perhaps at a wet or moldy grass eater that is eating kilograms, is not   
   converted past the first few grams of the get high dose, that way a bear, a   
   squirrel, a mouse, and a deer can get high off the same plant, fungi, or other   
   organism sourced    
   recreational drug even though they eat different amounts   
      
   It is possible different neurotransmitter neuron activations  at the nucleus   
   accumbens are differently enjoyable, they could have mice test out different   
   nucleus accumbens neuron and neurotransmitter particular effects and   
   combinations to find which have,   
    to my perception, tremendous enjoyment (fMRI of mice) with mating urge mate   
   presentation able to cause omitting repeat administration, it is beneficial if   
   the self administration of the recreational drug at nonhuman mammals is less   
   than human voluntary    
   repeat administration of caffeine, similar to the tremendously mild repeat use   
   effect at phenibut, while still being voluntarily and enjoyably used   
      
   Previously non edible plant parts, pine cones, palm tree bark, maple seeds,   
   sagebrush as well as others, could all have recreational drugs that nonhuman   
   (or humans) mammals enjoy that are harmless and where saliva fluidly and   
   enzymatically releases the    
   drug with 1-3 minutes at the mouth, chewing optional, and with an absence of   
   swallowing   
      
   It might be possible to have the living layer of tree bark, the cambium, be a   
   drug neutralizing chemical containing part of plants, then there would mostly   
   be an absence of harm to plants with tree bark at a variety of trees all over   
   the earth containing    
   recreational drugs that nonhuman mammals (as well as humans) enjoy, the   
   nonhuman mammal would omit nibbling the living part of the tree, to preserve   
   their enjoyable high, concentrating only on the nibbling the bark exterior   
      
   Longevity technology   
   Phenibut, a GABA active drug, reduces the appearance of inflammation at   
   dermatitis many double digit %, and to my perception causes more rapid   
   restoration of well dermis, I have read that inflammation is linked to non   
   youthful phenotype, testing phenibut    
   on mice to find out if it is longevizing as well as healthspan increasing, and   
   preserving of youthful phenotype is beneficial, if phenibut does cause   
   longevization as well as, or, greater healthspan then screening molecular   
   variations of phenibut to find    
   the most longevizing, healthspan increasing variations is a beneficial   
   longevity drug, notably there are also GABA active peptides, if screening a   
   library of these peptides finds longevizing as well as, or healthspan   
   increasing versions then gene therapy    
   that produces that peptide is a longevity technology and a one dose atopic   
   dermatitis curing or ameliorating treatment   
      
   What is the mechanism of phenibut reducing erythema, itch, and, notably,   
   rapidifying healing at dermatitis, the receptors and gene products of that   
   could be a basis for new antidermatitis drugs, possibly also antiinflammatory   
   longevity drugs, it is    
   possible GABA activity at the peripheral nervous system could be a part of   
   this effect, if variations on peripheral nervous system activity reduce   
   inflammation body wide, more than just at the dermis,  then they could find   
   out if peripheral nervous    
   system SNPs and allele variations have longevity effects at humans,   
   epigenetics, gene therapy, as well as new drugs that effect the peripheral   
   nervous system that are beneficial may be possible   
      
   AM or PM depot drug activation could be beneficial at administering drugs like   
   the longevity peptide AEDG epithalon, variation at body temperature sleep wake   
   temperature differences might be utilizable, gastrointestinal activity   
   differences,  pulse rate    
   average while asleep differences, these various differences combined, as well   
   as full side of body surface continuous contact difference from sleeping   
   (squishable polymer capsule) could all contribute to different am pm drug   
   administration    
      
   Longevity drug   
   Noting time of day, sleep wake cycle responsive drug administration from depot   
   injection could be possible it is possible some longevity drugs like AEDG   
   Epithalon, a pineal peptide have greater longevization activity when   
   repeatedly administered at just    
   part of the sleep wake cycle   
      
   --- SoupGate-Win32 v1.05   
    * Origin: you cannot sedate... all the things you hate (1:229/2)