From: treon3verdery@gmail.com   
      
   A different decanoic acid, that could also be screened for longevity effects   
   is an antimicrobial, “A recently developed biofilm inhibitor, cis-2-decenoic   
   acid (C2DA)”  if C2DA is like an order of magnitude or two more   
   antimicrobial than 10HDA, “   
   C2DA at concentrations of 500 μg/mL and above inhibited growth, while 125   
   μg/mL C2DA inhibited biofilm. Combination with antibiotics increased these   
   effects. At concentrations up to 500 μg/mL, there were no cytotoxic effects   
   on fibroblasts.”   
      
    it is possible it has other characteristics as well, and screening it for   
   longevity effects at c elegans could be beneficial   
      
      
      
   a 12 C fatty acid, that looks kind of like 10HDA that could be screened for   
   longevity effects is : “cis-2-decenoic acid appears to be functionally and   
   structurally related to the class of short-chain fatty acid signaling   
   molecules such as diffusible    
   signal factor which act as cell-to-cell communication molecules in bacteria   
   and fungi.”, “characterized a substituted fatty acid messenger,   
   cis-11-methyl-2-dodecenoic acid, called diffusible signal factor (DSF)”   
      
      
      
   also, “Treatments consisted of spent medium, CSM, cis-2-decenoic acid,   
   trans-2-decenoic acid, decanoic acid, and DSF at various concentrations.”,   
   “cis isomer of 2-decenoic acid was the organic compound” [that caused   
   biofilms to disperse], “The    
   compounds with the highest activity were two isomers of 2-decenoic acid. The   
   trans isomer (trans-2-decenoic acid) was shown by microtiter plate dispersion   
   bioassay to have activity only at millimolar concentrations, typically not low   
   enough to qualify as    
   a cell-cell signaling molecule. Figure Figure5B5B shows the dispersion   
   activity of increasing concentrations of cis-2-decenoic acid against biofilm   
   cultures of P. aeruginosa grown in microtiter plates. These results   
   demonstrated that the cis isomer (cis-   
   2-decenoic acid) was active over a concentration range from 1.0 nM to 10 mM”   
      
   There is some perspective here where a decanoic acid, of a slight variation   
   (cis rather than trans) is like an actual 1 million times more effective   
   (nanomoles compared with millimoles)at causing bacterial biofilms to detach;   
   so that brings up the    
   possibility, noting that DAEE causes greater happiness, better healing, and   
   other benefits at 2.9 mg/70Kg/24 hour human, that there could be a a 10HDA   
   version that has longevity effects at a couple orders of magnitude fewer atoms   
   per dose, or,    
   beneficially, has a longevity effect ten times larger than 27% (10HDA at   
   mice), notably salicylic acid causes yeast to live more than 400% longer, so   
   this could be possible; among organisms, a 720 minute longevity bug, compared   
   with a half century    
   duration termite queen is a a 36,500 times longevity difference, and at   
   mammals, a yearlong rodent compared with a 211 year whale is a 21100%   
   longevity difference; The antarctic sponge has a lifespan of 15550 years or   
   longer;   
      
      
      
   longevity technology:  10HDA at royal jelly is published as an HDACinhibitor,   
   causing acetylation which velocitizes and causes DNA transcription at   
   histones, I perceive I read a variety of HDACinhibitors have longevity and   
   wellness effects,  So, as a    
   beneficial complementary thing to making a bunch of 10HDA (or 10H2DA)   
   molecular variants and then screening them to find out if they cause greater   
   longevity at yeast, c elegans and 96 well plate vertebrate fish, and mammals,   
   they could make a bunch of    
   molecular variants of 10HDA that could be and quantified as to their   
   effectiveness as HDAC inhibitors, and noted as to their dose (mcg/ng)   
   effectiveness at causing HDAC inhibition at a variety of HDACs and   
   effectiveness at histones at cytostructures of a    
   variety of species, including humans, This gives all kinds of new molecular   
   modification possibilities of being effective:  10HDA dimers, halogenated   
   10HDA, 10HDA linked to a nuclear membrane transport peptide, 10HDA with a   
   butyrate (or propynate)    
   attached to it, or a valproate, basically any HDAC inhibitor molecule   
   partially modified to have a 10HDA on it,    As a molecular library to screen,   
   come up with a few hundred 10HDA variations all of which have a possibility of   
   being a better, faster,    
   stronger, more reaching of tissues that 10DHA might not usually reach HDAC   
   inhibitor, then screen the library of those hundreds of 10HDA molecule   
   variants, with things like multiwell plates with yeast, also c elegans can be   
   used to quantify better faster    
   stronger HDACinhibiting effects; Then at humans these versions might also   
   cause greater longevity at few mg or ug/dose   
      
      
      
      
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