From: treon3verdery@gmail.com   
      
    possible that without antibodies, this would cause autophagy at virus inected   
   cytes from their being at 30th percentile or less of wellness quantifications,   
   the incidental antiviral effect could be beneficial to both the person, and   
   even possibly reduce    
   the amount of viruses circulating at the population.  The 30th percentile of   
   wellness autophagy technology could be a nonimmunosystem new to me approach   
   that kind of incidentally removes virus infected, oncocyte, and even possibly,   
   fiberous nonutility    
   tissue, which might be replaced with weller cytes. It is kind of like a new to   
   me, multipurpose, new (30th percentile quantified effect at any nonoptimality)   
   kind of senolytic, with different criteria than various intracyte deleterious   
   products like    
   interleukins;   
      
      
      
   the versionbs of these technologies or others that create longevity, wellness,   
   and healthspan beneficial new versions of autophagyor germline enhancement or   
   optimization   
      
      
      
      
      
   Longevity technology: an antiglycation mechanism could be found at some   
   animals then tested at mice anfd humans to see if it increased longevity,   
   healthspan, and wellness, also, gene variations at humans like SNPs could be   
   correlated with wellness and    
   absence of or reduced glycation at humans, and then causality measured with   
   mouse studies to find beneficial genes for human gene enhancement or also   
   optimization; notably though I perceive plants also do the nonpreferred   
   glycation of proteins, so    
   anything at plants that is produced at cytes that reduces glycation could be   
   tested at the mammalian genome to find out if it benefits longevity, wellness,   
   and healthspan, notably the 40,000 year old King’s holly, the 10,000 year   
   old creosote bush, and    
   the 4k year old conifer might each have a different plant genetics of   
   antiglycation that benefits their longevity that could be tested at mice and   
   humans.   
      
      
      
   When a person gets gene therapy, they might like having a way to utilize their   
   previous genome at some or even all their cytes: backup with gene therapy:   
   crispr/cas9 appends the new genome to the previous genome, puts a start codon   
   (or start codon group)    
   imaginably at a sticks-out circly pouf on the nucleotide double lane topology,   
   the body ignores the first, previous, genome, which might even have some stop   
   codons crispr/cas9ed into it at easily recognized locations (sort of like   
   restriction enzymes say    
   “thing here” perhaps stop codons could be placed at the previous genome   
   anytime a CCCCC occured, so if editing it out it would be near errorless to   
   utilize the previous genome, if the utilizer felt like it.     
      
      
      
   I have not read about any siRNA longevity molecules, It is possible these are   
   possible, and that siRNA that heighten AMPK and decrease mTOR (or another 60%   
   greater mouse longevity mTOR drug, that works on just mTOR1 rather than mTOR1   
   and mTOR2), siRNA    
   might be even better at reaching the CNS through the blood brain barrier as   
   their AMU is less than some other nucleotides   
      
      
      
      
      
      
      
   I perveive there might be a million or more actin lanes per cyte, at 70   
   trillion cytes, that could be like a math iteration structure with a really   
   large number of math areas to model, algorithmize, and generate, something   
   like interpretations about    
   things as compared with, and possibly as a beneficial resource to the brain   
   and CNS;  Like what if the 70 trillion cytes with actin paths simulated   
   various effects of various possible things, and communicated the modelling   
   results with a one thing one    
   meaning language;   
      
      
      
   um, I perceive how DNA per cyte has lots more data space, it is just that   
   actin paths also have lots of functional movement, geometry, spatial   
   accessibility…   
      
      
      
   It likely already exists, but is there a CRISPR/cas9 automatic gene sequence   
   linker?  I perceive different lengths of DNA have different easiness of   
   transfection like 3/4 a decade ago (2011), but the perception I have of of   
   CRISPR/cas9 is that they have    
   figured out how to make. transfer, and activate things with out about 20,000   
   genes with simultaneous high velocity, high accuracy, and high editing sucess   
   (transfection); complementing that, perhaps at a variety of sizes, could be   
   something that is    
   effective at attaching one sequence to another, at a functional place and   
   physical form, (imaginably, histonated, less histonated, a loopy part   
   available because of a mitosis, translation as well as transcription event,   
   meiosis, or some new thing that is    
   new to me)   
      
      
      
   so, one approach is to find the easiest histones on earth;  some mammal has   
   histones with really long, super editable, physical like-new preservationess   
   above other mammalian histones, really available DNA; completely making a   
   synthetic sequence of that,    
   then making if even more genetic engineering friendly, then placing it at a   
   variety of mammals, likely including humans, could benefit DNA transcript   
   fidelity, DNA preservation, translation velocity at organisms, like humans, as   
   well as heightening    
   beneficial, functional, engineering friendly genetic editing, modification and   
   genetic engineering;    
      
      
      
   Also, besides unlooping things, and actually I have no idea what they do, but   
   I perceive DNA is unusually accessible during translation, mitosis, meiosis,   
   and possibly some kind of “make this” thing that something at the nucleus   
   says, like imaginably,   
    if something says “make ribosomes” perhaps hundreds of ribosome making   
   DNA locations get sequentially availabilized rather than just like one, over   
   and over again; so, it seems possible they have tried loading up a well human   
   cyte with a numerous    
   quantity of things to translate at DNA, so they could unspool a bunch of DNA,   
   efficiently, and edit it;   
      
      
      
   Along with making like a big list of DNA access producing translation   
   instructions, they might have some amazing thing like a DNA translation   
   smoothified new to me histone that makes DNA completely available to editing   
   (like crispr cas9 or more advanced)    
   while being a place to have a lot of DNA stay linear, functional, well,   
   effective, and immediately ok to utilize without repairs; the smoothified   
   histone could even be nifty at some ethynilization methylization   
   optionalizing, gene modification now able    
   to be unaffected from methylization and ethynilization molecular topology   
   effect; a smoothified histone like an inspection and upgrade access area of an   
   airplane;    
      
      
      
      
   [continued in next message]   
      
   --- SoupGate-Win32 v1.05   
    * Origin: you cannot sedate... all the things you hate (1:229/2)